G
Guest
Guest
But it's safe for you!
"If previous research on the subject was not enough, there is now undeniable evidence that suggests we should not be consuming aspartame at all in our diet. Switching over to sugar sweetened soda is also not a good alternative as this study also found that men consuming one or more sugar-sweetened sodas daily saw a 66 percent increase in non-Hodgkin lymphoma. It has become clear that having no soda at all in our diet is the ideal way to go. This would not only remove the aspartame and sugar risks, but it will also help in keeping your body in a more alkaline state"
http://www.collective-evolution.com/2012/12/09/aspartame-linked-leukemia-lymphoma-groundbreaking-study/
The Fear
A Wise Man (once sat next to him)
posted by steff on facebook.
France & Denmark have banned
it from the country...
RED BULL - slow death ...
Do NOT drink this drink anymore!
Pay attention...read everything...
As a public health safety, please pass on this email to all the contacts in your address book especially those with teenage children.
This drink is SOLD in all the supermarkets IN OUR country and our children ARE CONSUMING IT ON A TRIAL BASIS. IT can be FATAL.
RED BULL was created to stimulate the brains in people who are subjected to great physical force and in stress coma and never to be consumed like an innocent drink or soda pop.
RED BULL IS the energizer DRINK that is commercialized world-wide with its slogan: "It increases endurance, awakens the concentration capacity and the speed of reaction, offers more energy and improves the mood. All this can be found in a can of RED BULL, the power drink of the millennium."
RED BULL has managed to arrive in almost 100 countries worldwide. The RED BULL logo is targeted at young people and sportsmen, two attractive segments that have been captivated by the stimulus that the drink provides.
It was created by Dietrich Mateschitz, an industrialist of Austrian origin who discovered the drink by chance. It happened during a business trip to Hong Kong , when he was working at a factory that manufactured toothbrushes.
The liquid, based on a formula that contained caffeine and taurine, caused a rage in that country. Imagine the grand success of this drink in Europe where the product still did not exist, besides it was a superb opportunity to become an entrepreneur.
BUT THE TRUTH ABOUT THIS DRINK IS ANOTHER THING
FRANCE and DENMARK have just prohibited it as a cocktail of death, due to its vitamin components mixed with GLUCURONOLACTONE ' - a highly-dangerous chemical, which was developed by the United States Department of Defense during the sixties to stimulate the moral of the troops based in VIETNAM, which acted like a hallucinogenic drug that calmed the stress of the war.
But their effects in the organism were so devastating, that it was discontinued, because of the high index of cases of migraines, cerebral tumors and diseases of the liver that was evident in the soldiers who consumed it.
And in spite of it, in the can of RED BULL you can still find as one of its components: GLUCURONOLACTONE, categorized medically as a stimulant. But what it does not say on the can of RED BULL are the consequences of its consumption, and that has forced a series of WARNINGS...
1. It is dangerous to take it if you do not engage in physical exercise afterwards, since its energizing function accelerates the heart rate and can cause a sudden attack.
2. You run the risk of undergoing a cerebral hemorrhage, because RED BULL contains components that dilute the blood so that the heart utilizes less energy to pump the blood, and thus be able to deliver physical force with less effort being exerted.
3. It is prohibited to mix RED BULL with alcohol, because the mixture turns the drink into a " Deadly Bomb " that attacks the liver directly, causing the affected area never to regenerate anymore.
4. One of the main components of RED BULL is the B12 vitamin, used in medicine to recover patients who are in a coma ; from here the hypertension and the state of excitement which is experienced after taking it, as if you were in a drunken state.
5. The regular consumption of RED BULL triggers off symptoms in the form of a series of irreversible nervous and neuronal diseases.
CONCLUSION: It is a drink that should be prohibited in the entire world as when it is mixed with alcohol it creates a TIME BOMB for the human body, mainly between innocent adolescents and adults with little experience
France & Denmark have banned
it from the country...
RED BULL - slow death ...
Do NOT drink this drink anymore!
Pay attention...read everything...
As a public health safety, please pass on this email to all the contacts in your address book especially those with teenage children.
This drink is SOLD in all the supermarkets IN OUR country and our children ARE CONSUMING IT ON A TRIAL BASIS. IT can be FATAL.
RED BULL was created to stimulate the brains in people who are subjected to great physical force and in stress coma and never to be consumed like an innocent drink or soda pop.
RED BULL IS the energizer DRINK that is commercialized world-wide with its slogan: "It increases endurance, awakens the concentration capacity and the speed of reaction, offers more energy and improves the mood. All this can be found in a can of RED BULL, the power drink of the millennium."
RED BULL has managed to arrive in almost 100 countries worldwide. The RED BULL logo is targeted at young people and sportsmen, two attractive segments that have been captivated by the stimulus that the drink provides.
It was created by Dietrich Mateschitz, an industrialist of Austrian origin who discovered the drink by chance. It happened during a business trip to Hong Kong , when he was working at a factory that manufactured toothbrushes.
The liquid, based on a formula that contained caffeine and taurine, caused a rage in that country. Imagine the grand success of this drink in Europe where the product still did not exist, besides it was a superb opportunity to become an entrepreneur.
BUT THE TRUTH ABOUT THIS DRINK IS ANOTHER THING
FRANCE and DENMARK have just prohibited it as a cocktail of death, due to its vitamin components mixed with GLUCURONOLACTONE ' - a highly-dangerous chemical, which was developed by the United States Department of Defense during the sixties to stimulate the moral of the troops based in VIETNAM, which acted like a hallucinogenic drug that calmed the stress of the war.
But their effects in the organism were so devastating, that it was discontinued, because of the high index of cases of migraines, cerebral tumors and diseases of the liver that was evident in the soldiers who consumed it.
And in spite of it, in the can of RED BULL you can still find as one of its components: GLUCURONOLACTONE, categorized medically as a stimulant. But what it does not say on the can of RED BULL are the consequences of its consumption, and that has forced a series of WARNINGS...
1. It is dangerous to take it if you do not engage in physical exercise afterwards, since its energizing function accelerates the heart rate and can cause a sudden attack.
2. You run the risk of undergoing a cerebral hemorrhage, because RED BULL contains components that dilute the blood so that the heart utilizes less energy to pump the blood, and thus be able to deliver physical force with less effort being exerted.
3. It is prohibited to mix RED BULL with alcohol, because the mixture turns the drink into a " Deadly Bomb " that attacks the liver directly, causing the affected area never to regenerate anymore.
4. One of the main components of RED BULL is the B12 vitamin, used in medicine to recover patients who are in a coma ; from here the hypertension and the state of excitement which is experienced after taking it, as if you were in a drunken state.
5. The regular consumption of RED BULL triggers off symptoms in the form of a series of irreversible nervous and neuronal diseases.
CONCLUSION: It is a drink that should be prohibited in the entire world as when it is mixed with alcohol it creates a TIME BOMB for the human body, mainly between innocent adolescents and adults with little experience
The Fear
A Wise Man (once sat next to him)
http://en.wikipedia.org/wiki/Red_Bull
Health effects
Typical ingredients like caffeine, taurine and glucuronolactone have been assessed by health authorities for their safety. Health Canada conducted a review of the scientific literature on caffeine concluding that the general population of healthy adults is not at risk for potential adverse effects from caffeine if they limit their consumption to 400 mg per day.[18]
Taurine and glucuronolactone are normal body constituents and also naturally present in the human diet (e.g. scallops, fish, poultry and grain respectively). The European Food Safety Authority (EFSA) in its January 2009 opinion on the safety of energy drink ingredients concluded that the exposure to taurine and glucuronolactone at the levels presently used in energy drinks is not of safety concern.[19]
A review published in 2008 found no documented reports of negative or positive health effects associated with the amount of taurine used in energy drinks, including Red Bull. Caffeine and sugar levels in Red Bull are comparable to coffee and fruit juices, respectively.[20] Another publication found that "the number of available publications that really go into the details in this topic is also rather poor".[21]
[edit]
Caffeine
Main article: Caffeine
The caffeine of a single can of Red Bull is 80 mg/250 ml (32 mg/100 ml).[22][23] This is about the same as a normal coffee, or slightly less depending on the brewing method.[24] The actual caffeine level in Red Bull can vary depending on country, as some countries have legal restrictions on how much caffeine is allowed in drinks. As is the case with other caffeinated beverages, Red Bull drinkers may experience adverse effects as a result of overuse caffeine intoxication.
Energy drinks are not sports drinks and have not been formulated to deliver re-hydration. Water should be consumed in addition to energy drinks during exercise.
A 2008 position statement issued by the National Federation of State High School Associations made the following recommendations about general energy drink consumption for young athletes:[citation needed]
Water and appropriate sports drinks should be used for rehydration as outlined in the NFHS Document “Position Statement and Recommendations for Hydration to Minimize the Risk for Dehydration and Heat Illness”.
Energy drinks should not be used for hydration.
Information about the absence of benefit and the presence of potential risk associated with energy drinks should be widely shared among all individuals who interact with young athletes.
Energy drinks should not be consumed by athletes who are dehydrated.
Energy drinks should not be consumed, without prior medical approval, by athletes taking over the counter or prescription medications.
[edit]
Cardiovascular effects
A review of the scientific literature on caffeine concluded that moderate caffeine intake (less than 400 mg per day) does not adversely affect cardiovascular health.[25] A can of Red Bull, 250ml, contains 80 mg of caffeine which is less than a cup of coffee.[26]
There has been at least one case report of Red Bull overdose causing postural orthostatic tachycardia syndrome in a young athlete.[27] A February 3, 2009 article in The Daily Telegraph called, "Red Bull 'may have triggered heart condition that killed student' " reported the death of a 21-year-old woman who died after drinking four cans of Red Bull as well as alcohol at "social levels".[28] It was believed, but had not been proven, that she suffered from a rare heart condition called long QT Syndrome.[28] She was on medication for epilepsy and had an abnormally large heart. A medical examination found that there were no illegal drugs in her system. The article quoted a doctor as saying, "The coroner recorded that the 21-year-old woman died of natural causes."[2
Health effects
Typical ingredients like caffeine, taurine and glucuronolactone have been assessed by health authorities for their safety. Health Canada conducted a review of the scientific literature on caffeine concluding that the general population of healthy adults is not at risk for potential adverse effects from caffeine if they limit their consumption to 400 mg per day.[18]
Taurine and glucuronolactone are normal body constituents and also naturally present in the human diet (e.g. scallops, fish, poultry and grain respectively). The European Food Safety Authority (EFSA) in its January 2009 opinion on the safety of energy drink ingredients concluded that the exposure to taurine and glucuronolactone at the levels presently used in energy drinks is not of safety concern.[19]
A review published in 2008 found no documented reports of negative or positive health effects associated with the amount of taurine used in energy drinks, including Red Bull. Caffeine and sugar levels in Red Bull are comparable to coffee and fruit juices, respectively.[20] Another publication found that "the number of available publications that really go into the details in this topic is also rather poor".[21]
[edit]
Caffeine
Main article: Caffeine
The caffeine of a single can of Red Bull is 80 mg/250 ml (32 mg/100 ml).[22][23] This is about the same as a normal coffee, or slightly less depending on the brewing method.[24] The actual caffeine level in Red Bull can vary depending on country, as some countries have legal restrictions on how much caffeine is allowed in drinks. As is the case with other caffeinated beverages, Red Bull drinkers may experience adverse effects as a result of overuse caffeine intoxication.
Energy drinks are not sports drinks and have not been formulated to deliver re-hydration. Water should be consumed in addition to energy drinks during exercise.
A 2008 position statement issued by the National Federation of State High School Associations made the following recommendations about general energy drink consumption for young athletes:[citation needed]
Water and appropriate sports drinks should be used for rehydration as outlined in the NFHS Document “Position Statement and Recommendations for Hydration to Minimize the Risk for Dehydration and Heat Illness”.
Energy drinks should not be used for hydration.
Information about the absence of benefit and the presence of potential risk associated with energy drinks should be widely shared among all individuals who interact with young athletes.
Energy drinks should not be consumed by athletes who are dehydrated.
Energy drinks should not be consumed, without prior medical approval, by athletes taking over the counter or prescription medications.
[edit]
Cardiovascular effects
A review of the scientific literature on caffeine concluded that moderate caffeine intake (less than 400 mg per day) does not adversely affect cardiovascular health.[25] A can of Red Bull, 250ml, contains 80 mg of caffeine which is less than a cup of coffee.[26]
There has been at least one case report of Red Bull overdose causing postural orthostatic tachycardia syndrome in a young athlete.[27] A February 3, 2009 article in The Daily Telegraph called, "Red Bull 'may have triggered heart condition that killed student' " reported the death of a 21-year-old woman who died after drinking four cans of Red Bull as well as alcohol at "social levels".[28] It was believed, but had not been proven, that she suffered from a rare heart condition called long QT Syndrome.[28] She was on medication for epilepsy and had an abnormally large heart. A medical examination found that there were no illegal drugs in her system. The article quoted a doctor as saying, "The coroner recorded that the 21-year-old woman died of natural causes."[2
The Fear
A Wise Man (once sat next to him)
Glucuronolactone has received some notoriety due to urban legends that it was a Vietnam War-era drug manufactured by the American government. The rumor goes on to say that it was banned due to brain tumor-related deaths. The rumor has since been proven false, due to irreality of the cited British Medical Journal article and non-event "banning of its consumption". Furthermore, no warnings appear on the Food and Drug Administration website regarding its potential to cause brain tumors or other maladies.[2]
[edit]Uses
Glucuronolactone is rapidly absorbed and metabolized into non-toxic metabolites such as xylulose. In addition, humans may be able to use glucuronolactone as a precursor for ascorbic acid synthesis.[3] According to The Merck Index, it is also used as a detoxicant.[4]
Glucuronolactone is a popular ingredient in energy drinks with claims that it detoxifies the body. Although levels of glucuronolactone in energy drinks can far exceed those found in the rest of the diet, the European Food Safety Authority (EFSA) has concluded that exposure to glucuronolactone from regular consumption of energy drinks is not a safety concern. The no-observed-adverse-effect level of glucuronolactone is 1000 mg/kg/day.[5]
[edit]Uses
Glucuronolactone is rapidly absorbed and metabolized into non-toxic metabolites such as xylulose. In addition, humans may be able to use glucuronolactone as a precursor for ascorbic acid synthesis.[3] According to The Merck Index, it is also used as a detoxicant.[4]
Glucuronolactone is a popular ingredient in energy drinks with claims that it detoxifies the body. Although levels of glucuronolactone in energy drinks can far exceed those found in the rest of the diet, the European Food Safety Authority (EFSA) has concluded that exposure to glucuronolactone from regular consumption of energy drinks is not a safety concern. The no-observed-adverse-effect level of glucuronolactone is 1000 mg/kg/day.[5]
The Fear
A Wise Man (once sat next to him)
Saw this post on facebook
So apparently Pepsi are removing aspartame from their diet brands due to public opinion and demand.
However there is very little in the way of evidence to prove that aspartame causes health risks when taken in a moderate dose.
Here is a good table showing how many cans you would need to drink a day to hit the recommended 'safe' intake.... 17 cans anyone??
By the way, the safe intake is set 100 times lower than what is considered to be the toxic dose...... Thats 170 cans folks....
More worrying is the fact that the are replacing it with sucralose (US only), that has a safety limit less than 1/3rd that of aspartame.......
Want a diet coke?? Go for it.....
So apparently Pepsi are removing aspartame from their diet brands due to public opinion and demand.
However there is very little in the way of evidence to prove that aspartame causes health risks when taken in a moderate dose.
Here is a good table showing how many cans you would need to drink a day to hit the recommended 'safe' intake.... 17 cans anyone??
By the way, the safe intake is set 100 times lower than what is considered to be the toxic dose...... Thats 170 cans folks....
More worrying is the fact that the are replacing it with sucralose (US only), that has a safety limit less than 1/3rd that of aspartame.......
Want a diet coke?? Go for it.....
Attachments
The Fear
A Wise Man (once sat next to him)
Not sure Ryan, read a fair bit of compelling info on aspartame and it's side effects. Plenty of info online (and yes, you have to sift through the conspiracy and far fetched stuff online I realise!). What 'they' say is safe and what is safe are two different things, remember the trans fats that took years to be removed with food manufacturers denying any problem with them!?
39 mins · Like
*******
Jonathan, as said the 'safe' level here is 100 times below what is considered the toxic levels, so to get close to the toxic level you'd have to drink an unholy amount......
Particularly as they are replacing it in this instance with something that could be considered more toxic. No credible research exists showing issues in what would be considered a safe and reasonable dose.
32 mins · Like
*********
Sorry to spam your wall Ryan, but here's a good starting point for studies looking at the safety of aspartame...
5 mins · Like · 1
Thomas-Dobersen, D. (1989). Calculation of aspartame intake in children. Journal of the American Dietetic Association 89:831– 833.
Tilson, H.A., Hong, J.S., and Sobotka, T.J. (1991). High doses of as- partame have no effects on sensorimotor function or learning and memory in rats. Neurotoxicology and Teratology 13:27–35.
Tilson, H.A., Thai, L., Zhao, D., Sobotka, T.J., and Hong, J.S. (1989). Oral administration of aspartame is not proconvulsant in rats. Neu- rotoxicology 10:229–238.
Toledo, M.C., and Ioshi, S.H. (1995). Potential intake of intense sweet- eners in Brazil. Food Additives and Contaminants 12:799–808.
Tollefson, L. (1988). Monitoring adverse reactions to food additives in the U.S. Food and Drug Administration. Regulatory Toxicology and Pharmacology 8:438–446.
Tollefson, L., and Barnard, R.J. (1992). An analysis of FDA pas- sive surveillance reports of seizures associated with consumption of aspartame. Journal of the American Dietetic Association 92:598– 601.
Tollefson, L., Barnard, R.J., and Glinsmann, W.H. (1988). In Dietary Phenylalanine and Brain Function (R.J. Wurtman and E. Ritter- Walker, Eds.). Birkhauser Boston, Inc. New York, NY, between Ad- ministration and pp. 317–337.
Torii, K., Mimura, T., Takasaki, Y., and Ichimura, M. (1986). Di- etary aspartame with protein on plasma and brain amino acids, brain monoamines and behavior in rats. Physiology and Behavior 36:765– 771.
Trichopoulos, D. (1999). Aspartame and breast and other cancers. Re- sponse. Western Journal of Medicine 171:301.
Trutter, J.A., and Reno, F.E. (1973). SC-18862: Two-year toxicity study in the rat; Final report. E-33 and E-34. Hazleton Laboratories, Inc., Vienna, VA. Report P-T No. 838H71.
Tsakiris, S., Giannoulia-karantana, A., Simintzi, I., and Schulpis, K.H. (2006). The effect of aspartame metabolites on human erythrocyte membrane acetylcholinesterase activity. Pharmacological Research 53:1–5.
Tutelyan, V.A., Kravchenko, L.V., and Kuzmina, E.E. (1990). The ef- fect of aspartame on the activity of rat liver xenobiotic-metabolizing enzymes. Drug Metabolism and Disposition: The Biological Fate of Chemicals 18:223–225.
Vaclavik, V.A., and Christian, E.W. (2003). Essentials of Food Science. 2nd Edition. Kluwer Academic Publishers, New York. p. 27.
Van den Eeden, S.K., Koepsell, T.D., Longstreth, W.T., Jr., van Belle, G., Daling, J.R., and McKnight, B. (1994). Aspartame ingestion and headaches: A randomized crossover trial. Neurology 44:1787– 1793.
van der Laan, J., Lima, B.S., and Snodin, D. (2002). Alternatives mod- els in carcinogenicity testing—A European perspective. Toxicologic Pathology 30:157–159.
VCF (1999). Methanol. Database of Volatile Compounds in Food. TNO Nutrition and Food Research, Boelens Aroma Chemical Information Service, Zeist, The Netherlands. CD-ROM.
Vences-Mejia, A., Labra-Ruiz, N., Hernandez-Martinez, N., Dorado- Gonzalez, V., Gomez-Garduno, J., Perez-Lopez, I., Nosti-Palacios, R., Camacho Carranza, R., and Espinosa-Aguirre, J.J. (2006). The effect of aspartame on rat brain xenobiotic-metabolizing enzymes. Human & Experimental Toxicology 25:453–459.
Venkatachalam, S., Tyner, S.D., Pickering, C.R., Boley, S., Recio, L., French, J.E., and Donehower, L.A. (2001). Is p53 haploinsufficient for tumor suppression? Implications for the p53+/– mouse model in carcinogenicity testing. Toxicologic Pathology 29:147–154.
Virani, S.S., Mendoza, C.E., Ferreira, A.C., and de Marchena, E. (2003). Graves’ disease and pulmonary hypertension: Report of 2 cases. Texas Heart Institute Journal 30:314–315.
Wall, K.M. and Pardridge, W.M. (1990). Decreases in brain protein synthesis elicited by moderate increases in plasma phenylalanine. Biochemical and Biophysical Research Communications 168:1177– 1183.
Walton, R.G. (1988). The possible role of aspartame in seizure induc- tion, In Dietary Phenylalanine and Brain Function (R.J. Wurtman and E. Ritter-Walker, Eds.). Birkhauser Boston, Inc. New York, NY. pp. 159–162.
Walton, R.G., Hudak, R., and Green-Waite, R.J. (1993). Adverse reac- tions to aspartame: Double-blind challenge in patients from a vul- nerable population. Biological Psychiatry 34:13–17.
Wang, C.S. (1980). Induction of RNA C-type viruses by artificial sweet- eners. Scientific note. Proceedings of the National Science Council, Republic of China 4:424–427.
Ward, J.M., Rhem, S., and Reynolds, C.W. (1990). Tumors of the haematopoietic system. In Pathology of Tumors in Laboratory An- imals. Volume 1—Tumors of the Rat, Second Edition (V. Turusov and U. Mohr , Eds.). IARC Scientific Publications No. 99, Lyon, pp. 625–657.
Weihrauch, M.R., and Diehl, V. (2004). Artificial sweeteners—Do they bear a carcinogenic risk? Annals of Oncology 15:1460–1465.
Wolraich, M., Milich, R., Stumbo, P., and Schultz, F. (1985). Effects of sucrose ingestion on the behavior of hyperactive boys. Journal of Pediatrics 106:675–682.
Wolraich, M.L., Lindgren, S.D., Stumbo, P.J., Stegink, L.D., Appelbaum, M.I., and Kiritsy, M.C. (1994). Effects of diets high in sucrose or aspartame on the behavior and cognitive performance of children. New England Journal of Medicine 330:301–307.
Wurtman, R.J. (1983). Neurochemical changes following high-dose aspartame with dietary carbohydrates. New England Journal of Medicine 309:429–430.
Wurtman, R.J., and Maher, T.J. (1987). Effects of oral aspartame on plasma phenylalanine in humans and experimental rodents. Short note. Journal of Neural Transmission 70:169–173.
Wyss, C. (1993). Aspartame as a source of essential phenylalanine for the growth of oral anaerobes. FEMS Microbiology Letters 108:255– 258.
Xu, H., Staszewski, L., Tang, H., Adler, E., Zoller, M., and Li, X. (2004). Different functional roles of T1R subunits in the heteromeric taste receptors. Proceedings of the National Academy of Sciences of the United States of America 101:14258–14263.
Yokogoshi, H., Roberts, C.H., Caballero, B., and Wurtman, R.J. (1984). Effects of aspartame and glucose administration on brain and plasma
levels of large neutral amino acids and brain 5-hydroxyindoles. Amer-
ican Journal of Clinical Nutrition 40:1–7.
Yokogoshi, H., and Wurtman, R.J. (1986). Acute effects of oral or par-
enteral aspartame on catecholamine metabolism in various regions
of rat brain. Journal of Nutrition 116:356–364.
Yost, D.A. (1989). Clinical safety of aspartame. American Family
Physician 39:201–206.
Zametkin, A.J., Karoum, F., and Rapoport, J.L. (1987). Treatment of hy-
peractive children with D-phenylalanine. American Journal of Psy-
chiatry 144:792–794.
Zhang, C., Bordet, S., Karoum, F., and Commissiong, J.W. (1990).
Effect of precursors on the synthesis of catecholamines and on neu- rotransmission in the superior cervical ganglion of the rat. Journal of Neurochemistry 55:890–898.
Zhi, J.Q., and Levy, G. (1989). Aspartame and phenylalanine do not enhance theophylline-induced seizures in rats. Research Communi- cations in Chemical Pathology and Pharmacology 66:171–174.
5 mins · Like · 2
*****
Haha, that's quite extensive Nick
4 mins · Like · 1
Jonathan Fear Thing is though Ryan, I don't trust the food industry and their claims. They brought on obesity after-all with the low fat lie because the original report was battered by the powerful money in the sugar industry in the US. Hence the low fat products full of sugar, chemicals and additives to put back the taste taken out by the fat. The great lie! (Not that I'd eat processed ready meals anyway but many have and buy into the lie of these diet products)
When they say it is safe, I don't take it as said, that is for sure!
39 mins · Like
*******
Jonathan, as said the 'safe' level here is 100 times below what is considered the toxic levels, so to get close to the toxic level you'd have to drink an unholy amount......
Particularly as they are replacing it in this instance with something that could be considered more toxic. No credible research exists showing issues in what would be considered a safe and reasonable dose.
32 mins · Like
*********
Sorry to spam your wall Ryan, but here's a good starting point for studies looking at the safety of aspartame...
5 mins · Like · 1
Thomas-Dobersen, D. (1989). Calculation of aspartame intake in children. Journal of the American Dietetic Association 89:831– 833.
Tilson, H.A., Hong, J.S., and Sobotka, T.J. (1991). High doses of as- partame have no effects on sensorimotor function or learning and memory in rats. Neurotoxicology and Teratology 13:27–35.
Tilson, H.A., Thai, L., Zhao, D., Sobotka, T.J., and Hong, J.S. (1989). Oral administration of aspartame is not proconvulsant in rats. Neu- rotoxicology 10:229–238.
Toledo, M.C., and Ioshi, S.H. (1995). Potential intake of intense sweet- eners in Brazil. Food Additives and Contaminants 12:799–808.
Tollefson, L. (1988). Monitoring adverse reactions to food additives in the U.S. Food and Drug Administration. Regulatory Toxicology and Pharmacology 8:438–446.
Tollefson, L., and Barnard, R.J. (1992). An analysis of FDA pas- sive surveillance reports of seizures associated with consumption of aspartame. Journal of the American Dietetic Association 92:598– 601.
Tollefson, L., Barnard, R.J., and Glinsmann, W.H. (1988). In Dietary Phenylalanine and Brain Function (R.J. Wurtman and E. Ritter- Walker, Eds.). Birkhauser Boston, Inc. New York, NY, between Ad- ministration and pp. 317–337.
Torii, K., Mimura, T., Takasaki, Y., and Ichimura, M. (1986). Di- etary aspartame with protein on plasma and brain amino acids, brain monoamines and behavior in rats. Physiology and Behavior 36:765– 771.
Trichopoulos, D. (1999). Aspartame and breast and other cancers. Re- sponse. Western Journal of Medicine 171:301.
Trutter, J.A., and Reno, F.E. (1973). SC-18862: Two-year toxicity study in the rat; Final report. E-33 and E-34. Hazleton Laboratories, Inc., Vienna, VA. Report P-T No. 838H71.
Tsakiris, S., Giannoulia-karantana, A., Simintzi, I., and Schulpis, K.H. (2006). The effect of aspartame metabolites on human erythrocyte membrane acetylcholinesterase activity. Pharmacological Research 53:1–5.
Tutelyan, V.A., Kravchenko, L.V., and Kuzmina, E.E. (1990). The ef- fect of aspartame on the activity of rat liver xenobiotic-metabolizing enzymes. Drug Metabolism and Disposition: The Biological Fate of Chemicals 18:223–225.
Vaclavik, V.A., and Christian, E.W. (2003). Essentials of Food Science. 2nd Edition. Kluwer Academic Publishers, New York. p. 27.
Van den Eeden, S.K., Koepsell, T.D., Longstreth, W.T., Jr., van Belle, G., Daling, J.R., and McKnight, B. (1994). Aspartame ingestion and headaches: A randomized crossover trial. Neurology 44:1787– 1793.
van der Laan, J., Lima, B.S., and Snodin, D. (2002). Alternatives mod- els in carcinogenicity testing—A European perspective. Toxicologic Pathology 30:157–159.
VCF (1999). Methanol. Database of Volatile Compounds in Food. TNO Nutrition and Food Research, Boelens Aroma Chemical Information Service, Zeist, The Netherlands. CD-ROM.
Vences-Mejia, A., Labra-Ruiz, N., Hernandez-Martinez, N., Dorado- Gonzalez, V., Gomez-Garduno, J., Perez-Lopez, I., Nosti-Palacios, R., Camacho Carranza, R., and Espinosa-Aguirre, J.J. (2006). The effect of aspartame on rat brain xenobiotic-metabolizing enzymes. Human & Experimental Toxicology 25:453–459.
Venkatachalam, S., Tyner, S.D., Pickering, C.R., Boley, S., Recio, L., French, J.E., and Donehower, L.A. (2001). Is p53 haploinsufficient for tumor suppression? Implications for the p53+/– mouse model in carcinogenicity testing. Toxicologic Pathology 29:147–154.
Virani, S.S., Mendoza, C.E., Ferreira, A.C., and de Marchena, E. (2003). Graves’ disease and pulmonary hypertension: Report of 2 cases. Texas Heart Institute Journal 30:314–315.
Wall, K.M. and Pardridge, W.M. (1990). Decreases in brain protein synthesis elicited by moderate increases in plasma phenylalanine. Biochemical and Biophysical Research Communications 168:1177– 1183.
Walton, R.G. (1988). The possible role of aspartame in seizure induc- tion, In Dietary Phenylalanine and Brain Function (R.J. Wurtman and E. Ritter-Walker, Eds.). Birkhauser Boston, Inc. New York, NY. pp. 159–162.
Walton, R.G., Hudak, R., and Green-Waite, R.J. (1993). Adverse reac- tions to aspartame: Double-blind challenge in patients from a vul- nerable population. Biological Psychiatry 34:13–17.
Wang, C.S. (1980). Induction of RNA C-type viruses by artificial sweet- eners. Scientific note. Proceedings of the National Science Council, Republic of China 4:424–427.
Ward, J.M., Rhem, S., and Reynolds, C.W. (1990). Tumors of the haematopoietic system. In Pathology of Tumors in Laboratory An- imals. Volume 1—Tumors of the Rat, Second Edition (V. Turusov and U. Mohr , Eds.). IARC Scientific Publications No. 99, Lyon, pp. 625–657.
Weihrauch, M.R., and Diehl, V. (2004). Artificial sweeteners—Do they bear a carcinogenic risk? Annals of Oncology 15:1460–1465.
Wolraich, M., Milich, R., Stumbo, P., and Schultz, F. (1985). Effects of sucrose ingestion on the behavior of hyperactive boys. Journal of Pediatrics 106:675–682.
Wolraich, M.L., Lindgren, S.D., Stumbo, P.J., Stegink, L.D., Appelbaum, M.I., and Kiritsy, M.C. (1994). Effects of diets high in sucrose or aspartame on the behavior and cognitive performance of children. New England Journal of Medicine 330:301–307.
Wurtman, R.J. (1983). Neurochemical changes following high-dose aspartame with dietary carbohydrates. New England Journal of Medicine 309:429–430.
Wurtman, R.J., and Maher, T.J. (1987). Effects of oral aspartame on plasma phenylalanine in humans and experimental rodents. Short note. Journal of Neural Transmission 70:169–173.
Wyss, C. (1993). Aspartame as a source of essential phenylalanine for the growth of oral anaerobes. FEMS Microbiology Letters 108:255– 258.
Xu, H., Staszewski, L., Tang, H., Adler, E., Zoller, M., and Li, X. (2004). Different functional roles of T1R subunits in the heteromeric taste receptors. Proceedings of the National Academy of Sciences of the United States of America 101:14258–14263.
Yokogoshi, H., Roberts, C.H., Caballero, B., and Wurtman, R.J. (1984). Effects of aspartame and glucose administration on brain and plasma
levels of large neutral amino acids and brain 5-hydroxyindoles. Amer-
ican Journal of Clinical Nutrition 40:1–7.
Yokogoshi, H., and Wurtman, R.J. (1986). Acute effects of oral or par-
enteral aspartame on catecholamine metabolism in various regions
of rat brain. Journal of Nutrition 116:356–364.
Yost, D.A. (1989). Clinical safety of aspartame. American Family
Physician 39:201–206.
Zametkin, A.J., Karoum, F., and Rapoport, J.L. (1987). Treatment of hy-
peractive children with D-phenylalanine. American Journal of Psy-
chiatry 144:792–794.
Zhang, C., Bordet, S., Karoum, F., and Commissiong, J.W. (1990).
Effect of precursors on the synthesis of catecholamines and on neu- rotransmission in the superior cervical ganglion of the rat. Journal of Neurochemistry 55:890–898.
Zhi, J.Q., and Levy, G. (1989). Aspartame and phenylalanine do not enhance theophylline-induced seizures in rats. Research Communi- cations in Chemical Pathology and Pharmacology 66:171–174.
5 mins · Like · 2
*****
Haha, that's quite extensive Nick
4 mins · Like · 1
Jonathan Fear Thing is though Ryan, I don't trust the food industry and their claims. They brought on obesity after-all with the low fat lie because the original report was battered by the powerful money in the sugar industry in the US. Hence the low fat products full of sugar, chemicals and additives to put back the taste taken out by the fat. The great lie! (Not that I'd eat processed ready meals anyway but many have and buy into the lie of these diet products)
When they say it is safe, I don't take it as said, that is for sure!
The Fear
A Wise Man (once sat next to him)
Is aspartame bad for you? The artificial sweetener, used in a huge variety of diet products from soft drinks to yoghurts, chewing gum to frozen desserts, is being dropped by Diet Pepsi in the US. “Aspartame is the number one reason consumers are dropping diet soda,” says Seth Kaufman, vice-president of Pepsi, whose sales of diet soda dropped 5% in the US last year. But why?
http://www.theguardian.com/world/shortcuts/2015/apr/28/diet-pepsi-dropped-aspatame-in-us-is-artificial-sweetener-dangerous
There have been large-scale randomised trials and cohort studies investigating it, including those undertaken by the US National Cancer Institute and the European Food Safety Authority – all of which concluded aspartame is safe in moderate doses.
Yet it has been linked to multiple sclerosis, lupus, brain tumours, blindness, seizures, mental health problems and birth defects, probably because its components can, in high enough doses, be harmful. But aspartame doesn’t contain high quantities of aspartic acid (also found in avocados and asparagus), or methanol (found in beer, wine and tomatoes). Phenylalanine is harmful, but only to the unlucky few with phenylketonuria, a rare genetic disorder. For everyone else, aspartame is safe. The daily recommended dose is 40mg per kg of body weight. For perspective, you would have to drink 12 cans of Diet Pepsi to hit that.
http://www.theguardian.com/world/shortcuts/2015/apr/28/diet-pepsi-dropped-aspatame-in-us-is-artificial-sweetener-dangerous
There have been large-scale randomised trials and cohort studies investigating it, including those undertaken by the US National Cancer Institute and the European Food Safety Authority – all of which concluded aspartame is safe in moderate doses.
Yet it has been linked to multiple sclerosis, lupus, brain tumours, blindness, seizures, mental health problems and birth defects, probably because its components can, in high enough doses, be harmful. But aspartame doesn’t contain high quantities of aspartic acid (also found in avocados and asparagus), or methanol (found in beer, wine and tomatoes). Phenylalanine is harmful, but only to the unlucky few with phenylketonuria, a rare genetic disorder. For everyone else, aspartame is safe. The daily recommended dose is 40mg per kg of body weight. For perspective, you would have to drink 12 cans of Diet Pepsi to hit that.
G
Guest
Guest
I don't trust them either claiming x amount of doses is ok for you doooon't you worry,,,feck off i don't want any aspertame in anything me or my kids consume thank you very much, i suppose a dab of sionide here oh and whilst yam at it go add some other chemical sh*t that burns the insides out,,,cheaky ba**ads,
Makes me cringe reading this sh*t cos when i was younger in my teens i drank between 2 litre bottles of coke sometimes on a weekend i would get through loads of them, my kids am happy with fruit juices no fizzy crap.
And it does make you wonder what in modern society these chemicals added to societies diet intake problems they have really caused.
Makes me cringe reading this sh*t cos when i was younger in my teens i drank between 2 litre bottles of coke sometimes on a weekend i would get through loads of them, my kids am happy with fruit juices no fizzy crap.
And it does make you wonder what in modern society these chemicals added to societies diet intake problems they have really caused.
G
Guest
Guest
Are true, damn it so i can't blame the ba**ads for the way i am pfffff
Juan Mourep
Vital 1st Team Regular
When they say you'd have to drink x amount of cans, they are trying to distract you from the fact that when it is in so much food/drink/medicines/mouthwash it is quite easy to get a very high dose throughout the day.
If it's so safe why do you have to wear protective gear and breathing apparatus when you are dealing with it?
If it's so safe why do you have to wear protective gear and breathing apparatus when you are dealing with it?
